By John R. Baker, Ralph Muller, David Rollinson

ISBN-10: 0120317583

ISBN-13: 9780120317585

The Advances in Parasitology sequence includes in-depth reports on present issues of curiosity in modern parasitology. It contains clinical experiences on parasites of significant impression, corresponding to trypanosomiasis and scabies, and extra conventional components, corresponding to zoology, taxonomy, and lifestyles historical past, which form present considering and purposes. * sequence has the second one optimum ISI influence consider the parasitology workforce! (4.818 in 2002) * individuals are overseas specialists within the box

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1999). 2. THE MÈS AS HOST CELLS FOR LEISHMANIA SPP. 1. Binding and Internalization of Promastigotes and Amastigotes Both promastigotes and amastigotes can be phagocytosed by MÈs through an actin-mediated phagocytic process. Recent studies show that in most cases, phagocytosis of both L. major and L. amazonensis metacyclic promastigotes proceeds first by the engulfment of the parasite cell body followed by the progressive internalization of the flagellum. , 2002; N. Courret, S. Henri and E. Handman, unpublished observation).

Non replicative amastigotes NO synthase 2 NO CD40 NADPH oxidase activation inhibition MIF TNFTNF-α ILIL-7 IFNIFN-α/β IFN-γ Figure 6 Mechanisms of Leishmania killing developed by MÈs and how parasites can evade them. Several cytokines produced by MÈs themselves (bent arrows in the lower part of the figure) or by other cells such as effector CD4 Th1 and CD8 T lymphocytes can activate or induce several leishmanicidal mechanisms (black arrows) while other cytokines synthesized by MÈs (bent arrows in the upper part of the figure) or by other cells such as CD4 Th2 lymphocytes can block the activation or the induction of these same mechanisms (black T).

3). c Invariant chains involved in the trafficking of MHC class II molecules. d MÈs treated with IFN-
. e Not determined. , 2001, 2003). The origin of the discrepancy between the results of Courret et al. and the latter ones is unclear, but the fact that different Leishmania strains and host cells were used in these studies is an important point to consider. In any case, the generality of the model proposed by M. Desjardins et al. was recently questioned by several new findings. First, it was found that, at least for L.

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Advances in Parasitology, Vol. 58 by John R. Baker, Ralph Muller, David Rollinson


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